Cell death induced by the ER stressor thapsigargin involves death receptor 5, a non-autophagic function of MAP1LC3B, and distinct contributions from unfolded protein response components

Research output: Contribution to journalJournal articlepeer-review

Documents

Original languageEnglish
Article number12
JournalCell Communication and Signaling
Volume18
Number of pages23
ISSN1478-811X
DOIs
Publication statusPublished - 2020

    Research areas

  • Thapsigargin, SERCA, Unfolded protein response, DR5, Caspase-8, PERK, ATF4, CHOP, IRE1, XBP1s, JNK, LC3B, Cell death, Apoptosis, Autophagy

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ID: 240009184