Ligands for the GABAA receptor complex

Research output: Chapter in Book/Report/Conference proceedingBook chapterResearchpeer-review

Standard

Ligands for the GABAA receptor complex. / Krogsgaard-Larsen, Povl; Frølund, Bente; Kristiansen, Uffe; Ebert, Bjarke.

Glutamate and GABA Receptors and Transporters: Structure, Function and Pharmacology. CRC Press, 2001. p. 236-274.

Research output: Chapter in Book/Report/Conference proceedingBook chapterResearchpeer-review

Harvard

Krogsgaard-Larsen, P, Frølund, B, Kristiansen, U & Ebert, B 2001, Ligands for the GABAA receptor complex. in Glutamate and GABA Receptors and Transporters: Structure, Function and Pharmacology. CRC Press, pp. 236-274.

APA

Krogsgaard-Larsen, P., Frølund, B., Kristiansen, U., & Ebert, B. (2001). Ligands for the GABAA receptor complex. In Glutamate and GABA Receptors and Transporters: Structure, Function and Pharmacology (pp. 236-274). CRC Press.

Vancouver

Krogsgaard-Larsen P, Frølund B, Kristiansen U, Ebert B. Ligands for the GABAA receptor complex. In Glutamate and GABA Receptors and Transporters: Structure, Function and Pharmacology. CRC Press. 2001. p. 236-274

Author

Krogsgaard-Larsen, Povl ; Frølund, Bente ; Kristiansen, Uffe ; Ebert, Bjarke. / Ligands for the GABAA receptor complex. Glutamate and GABA Receptors and Transporters: Structure, Function and Pharmacology. CRC Press, 2001. pp. 236-274

Bibtex

@inbook{88eb1f71b56d4a88a941293758cc7ee2,
title = "Ligands for the GABAA receptor complex",
abstract = "The neutral amino acid, 4-aminobutanoic acid (GABA), is an inhibitory transmitter in the central nervous system (CNS). Furthermore, GABA is involved as a neurotransmitter and/or a paracrine effector in the regulation of a variety of physiological mechanisms in the periphery. Some of these latter functions may be under central GABA control, whereas others are managed by local GABA neurons. A large percentage, perhaps the majority, of central neurons are under GABA control. The complex mechanisms underlying the GABA-mediated neurotransmission have been extensively studied using a broad spectrum of electrophysiological, neurochemical, pharmacological, and, in recent years, molecular biological techniques (Curtis and Johnston, 1974; Krnjevic, 1974; Olsen and Venter, 1986; Redburn and Schousboe, 1987; Bowery and Nistico, 1989; Biggio and Costa, 1990; Bowery et al., 1990; Schousboe et al., 1992a; Doble and Martin, 1996; Krogsgaard-Larsen et al., 1997).",
author = "Povl Krogsgaard-Larsen and Bente Fr{\o}lund and Uffe Kristiansen and Bjarke Ebert",
note = "Publisher Copyright: {\textcopyright} 2001 by Taylor & Francis Group, LLC.",
year = "2001",
month = jan,
day = "1",
language = "English",
isbn = "9780748408818",
pages = "236--274",
booktitle = "Glutamate and GABA Receptors and Transporters",
publisher = "CRC Press",

}

RIS

TY - CHAP

T1 - Ligands for the GABAA receptor complex

AU - Krogsgaard-Larsen, Povl

AU - Frølund, Bente

AU - Kristiansen, Uffe

AU - Ebert, Bjarke

N1 - Publisher Copyright: © 2001 by Taylor & Francis Group, LLC.

PY - 2001/1/1

Y1 - 2001/1/1

N2 - The neutral amino acid, 4-aminobutanoic acid (GABA), is an inhibitory transmitter in the central nervous system (CNS). Furthermore, GABA is involved as a neurotransmitter and/or a paracrine effector in the regulation of a variety of physiological mechanisms in the periphery. Some of these latter functions may be under central GABA control, whereas others are managed by local GABA neurons. A large percentage, perhaps the majority, of central neurons are under GABA control. The complex mechanisms underlying the GABA-mediated neurotransmission have been extensively studied using a broad spectrum of electrophysiological, neurochemical, pharmacological, and, in recent years, molecular biological techniques (Curtis and Johnston, 1974; Krnjevic, 1974; Olsen and Venter, 1986; Redburn and Schousboe, 1987; Bowery and Nistico, 1989; Biggio and Costa, 1990; Bowery et al., 1990; Schousboe et al., 1992a; Doble and Martin, 1996; Krogsgaard-Larsen et al., 1997).

AB - The neutral amino acid, 4-aminobutanoic acid (GABA), is an inhibitory transmitter in the central nervous system (CNS). Furthermore, GABA is involved as a neurotransmitter and/or a paracrine effector in the regulation of a variety of physiological mechanisms in the periphery. Some of these latter functions may be under central GABA control, whereas others are managed by local GABA neurons. A large percentage, perhaps the majority, of central neurons are under GABA control. The complex mechanisms underlying the GABA-mediated neurotransmission have been extensively studied using a broad spectrum of electrophysiological, neurochemical, pharmacological, and, in recent years, molecular biological techniques (Curtis and Johnston, 1974; Krnjevic, 1974; Olsen and Venter, 1986; Redburn and Schousboe, 1987; Bowery and Nistico, 1989; Biggio and Costa, 1990; Bowery et al., 1990; Schousboe et al., 1992a; Doble and Martin, 1996; Krogsgaard-Larsen et al., 1997).

UR - http://www.scopus.com/inward/record.url?scp=84862431049&partnerID=8YFLogxK

M3 - Book chapter

AN - SCOPUS:84862431049

SN - 9780748408818

SP - 236

EP - 274

BT - Glutamate and GABA Receptors and Transporters

PB - CRC Press

ER -

ID: 312029340